the disease is linked to an imbalance of the intestinal flora

New proof of the close relationship between the health of our intestinal flora and that of our brain, researchers have shown that Alzheimer’s disease is indeed linked to an imbalance in our microbiota.

The development of Alzheimer’s disease is well linked to a specific imbalance of the intestinal microbiota. This confirmation comes to us from Switzerland and Italy but several studies had already noted this correlation between the degenerative disease and our intestinal flora.

In this study, published on November 10 in the Journal of Alzheimer’s Disease, scientists have in particular demonstrated an inflammatory phenomenon detected in the blood of people suffering from Alzheimer’s disease. By studying 89 people aged 65 to 85, some with the disease and others not, they showed that this inflammation in patients could act as a “mediator” between the microbriot and the brain.

Soon a cocktail to rebalance the microbiota and prevent Alzheimer’s disease?
These results are important in research against Alzheimer’s disease. They allow, specifies a press release from the University of Geneva, “to consider new preventive strategies based on the modulation of the microbiota of people at risk. However, “ taking a cocktail to restore the balance of the intestinal microbiota or products to feed the good bacteria would only be effective at a very early stage of the disease “. It therefore remains to develop protocols making it possible to identify people at high risk in order to treat them well before the appearance of detectable symptoms ”.

This is not the first time that the link between microbiota and Alzheimer’s disease has been studied. Other research had shown an alteration in the microbiota of patients or a high level of inflammatory intestinal bacteria. However, proteins produced by these bacteria are suspected of modifying the interaction between the immune system and the nervous system.

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Each year, 225,000 new Alzheimer’s patients are detected in France. While the origin of the disease is still unknown, considerable progress has been made, particularly in the progression of lesions in the heart of the brain. They cause memory loss, language disorders, mood swings, dexterity and recognition disorders, as well as depression, sleep and appetite disturbances, delusions …

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