A predawn collision involving a truck transporting heavy equipment forced the Jakarta Public Works Office to dismantle a pedestrian bridge (JPO) in South Jakarta’s Tendean area on July 14. The structure was deemed a public safety risk after a technical inspection revealed severe damage to the main framework.
Shifted Columns and Structural Failure
The impact was not superficial. According to officials, the collision shifted a support column and dislodged several structural joints, compromising the bridge’s integrity. The risk of a spontaneous collapse in a high-traffic urban corridor left the Jakarta Provincial Government with little choice: the entire facility had to be demolished.
The Dismantling of Jalan Tendean
Personnel from multiple agencies executed the removal in stages. Using heavy equipment, crews tore down the structure while authorities secured the zone with safety barriers. To prevent total gridlock along Jalan Tendean, traffic engineering measures were implemented to maintain vehicle flow during the operation.

Mitigating Risk Over Repair
Assessing a Potential Replacement
While the area is cleared, the government has urged the public to follow officer instructions and utilize alternative routes. The future of the Tendean crossing remains under review; authorities will assess whether a new pedestrian bridge is needed at the same location once demolition is complete.
If a replacement is commissioned, officials state it will be designed in accordance with current construction safety standards to provide a safer crossing and ensure smooth traffic flow in the area.
Quick Facts: The Tendean Collision
Why was the bridge dismantled?
A truck carrying heavy equipment struck the structure, causing damage that a technical inspection deemed unsafe for public use.
What was the specific damage?
The impact shifted one support column out of position and dislodged several structural joints.
Will it be rebuilt?
Authorities will assess the need for a new bridge after demolition, ensuring any new structure meets current safety standards.
Worth a look
- Young Man Stabbed in Oslo: One Arrested
- IDF and Shin Bet Kill Hamas Terrorists in Gaza Strip
- Breakthrough Salk Study Uncovers Mechanism Behind Immunotherapy Resistance: Interferons, Mitochondrial Dysfunction, and PGE2″ Interferons, mitochondrial dysfunction and PGE2: Salk study reveals mechanism behind immunotherapy resistance. Boost its search engine visibility with relevant keywords for maximum impact. Immunotherapy resistance remains one of the biggest hurdles in cancer treatment. According to a recent study published in the journal Nature Communications, scientists at the Salk Institute have made a groundbreaking discovery that sheds light on the underlying mechanisms behind this resistance. The study reveals that interferons, a type of protein that plays a crucial role in the immune system, can contribute to mitochondrial dysfunction in cancer cells. This dysfunction can lead to the production of prostaglandin E2 (PGE2), a molecule that promotes tumor growth and resistance to immunotherapy. In their study, the researchers found that PGE2 production was a key factor in the development of immunotherapy resistance in cancer cells. The team used a combination of experimental and computational models to investigate the relationship between interferons, mitochondrial dysfunction, and PGE2 production. The findings of the study suggest that targeting PGE2 production could be a potential strategy for overcoming immunotherapy resistance. The researchers propose that blocking PGE2 receptors or inhibiting its production could help restore the function of mitochondria in cancer cells, making them more susceptible to immunotherapy. The study’s authors hope that their findings will pave the way for the development of new therapies that can overcome immunotherapy resistance and improve treatment outcomes for cancer patients. Key Takeaways: – Interferons contribute to mitochondrial dysfunction in cancer cells – Mitochondrial dysfunction leads to PGE2 production, promoting tumor growth and resistance to immunotherapy – Targeting PGE2 production could be a potential strategy for overcoming immunotherapy resistance – Restoring mitochondrial function in cancer cells could make them more susceptible to immunotherapy Keywords: immunotherapy resistance, interferons, mitochondrial dysfunction, PGE2, Salk Institute, cancer treatment, breakthrough study, Nature Communications. (archyworldys.com)